 |
|
What, If Anything, Is Spinal Shock?
Wilhelm Nacimiento, MD;
Johannes Noth, MD
Arch Neurol. 1999;56:1033-1035.
|
|
| Since this article does not have an abstract, we have provided the first 150 words of the full text and any section headings. |
|
|
|
The term "spinal shock" describes the sudden and transient suppression of neural functions below the level of acute spinal cord lesions, usually in the setting of trauma, ischemia, hemorrhage, or inflammatory diseases. The syndrome of spinal shock primarily refers to motor and autonomic disorders (see Young and Woolsey1 and Guttmann2 for reviews) mediated by the reduced excitability of spinal neurons following disconnection from descending input.3 During this state, a flaccid paralysis of skeletal muscles occurs, and all tendon, cutaneous, and autonomic reflexes integrated in the spinal cord below the lesion are abolished or greatly reduced. Following variable periods of time, from several days up to 4 to 6 weeks, spinal reflexes return and eventually become exaggerated as the syndrome of spasticity develops (see Noth4 for review).
The pathophysiology of spinal shock and spasticity is thought to involve neuronal mechanisms of inhibition and reorganization, respectively, . . . [Full Text of this Article] MARSHALL HALL, 1841
SIR CHARLES SCOTT SHERRINGTON, 1897
SIR CHARLES SCOTT SHERRINGTON, 1947
GRAYSON McCOUCH, 1971
SIR LUDWIG GUTTMANN, 1976
CONCLUSIONS
Department of Neurology, Aachen University Medical School, Aachen, Germany.
THIS ARTICLE HAS BEEN CITED BY OTHER ARTICLES
Immediate Changes in Tuning of Inferior Colliculus Neurons Following Acute Lesions of Cat Spiral Ganglion
Snyder and Sinex
J. Neurophysiol. 2002;87:434-452.
ABSTRACT
| FULL TEXT
|
