You are seeing this message because your Web browser does not support basic Web standards. Find out more about why this message is appearing and what you can do to make your experience on this site better.


ABOUT ARCHIVES
Advanced Search

Welcome   | My Account | E-mail Alerts | Access Rights | Sign In


  Vol. 56 No. 10, October 1999 TABLE OF CONTENTS
  Archives
  •  Online Features
  Editorial
 This Article
 •Full text
 •PDF
 • Reply to article
 •Send to a friend
 • Save in My Folder
 •Save to citation manager
 •Permissions
 Citing Articles
 •Citing articles on Web of Science (5)
 •Contact me when this article is cited
 Related Content
 •Related article
 •Similar articles in this journal
 Topic Collections
 •Neurology
 •Neurology, Other
 •Alert me on articles by topic
 Social Bookmarking
  Add to CiteULike Add to Connotea Add to Del.icio.us Add to Digg Add to Reddit Add to Technorati Add to Twitter What's this?

Ventricular Volume and Transmural Pressure Gradient in Normal Pressure Hydrocephalus

Arch Neurol. 1999;56:1199-1200.

Since this article does not have an abstract, we have provided the first 150 words of the full text and any section headings.

NORMAL PRESSURE hydrocephalus (NPH) is characterized by the clinical triad of gait disorder, cognitive dysfunction, and incontinence associated with ventriculomegaly and normal cerebrospinal fluid (CSF) opening pressure.1-2 This syndrome is difficult to differentiate from subcortical arteriosclerotic encephalopathy, which is far more commonly the source of the clinical triad. Although ventricular shunting of NPH is effective in halting progression and reversing deficits in some patients, treatment failures abound. Treatment failures are in part attributable to faulty selection of patients and/or suboptimal shunting techniques. In this issue of the ARCHIVES, Bergsneider et al3 help to refocus attention on some of the important issues in treatment of adult patients with NPH.

Normal pressure hydrocephalus was recently the subject of fine reviews by Vanneste et al,4-5 so only the briefest overview is needed here. It may be communicating or noncommunicating. The noncommunicating type of NPH is most commonly caused by aqueductal stenosis. Approximately 50% . . . [Full Text of this Article]



Add to CiteULike CiteULike   Add to Connotea Connotea   Add to Del.icio.us Del.icio.us   Add to Digg Digg   Add to Reddit Reddit   Add to Technorati Technorati   Add to Twitter Twitter     What's this?

RELATED ARTICLE

Beneficial Effect of Siphoning in Treatment of Adult Hydrocephalus
Marvin Bergsneider, Warwick J. Peacock, John C. Mazziotta, and Donald P. Becker
Arch Neurol. 1999;56(10):1224-1229.
ABSTRACT | FULL TEXT  






HOME | CURRENT ISSUE | PAST ISSUES | TOPIC COLLECTIONS | CME | SUBMIT | SUBSCRIBE | HELP
CONDITIONS OF USE | PRIVACY POLICY | CONTACT US | SITE MAP
 
© 1999 American Medical Association. All Rights Reserved.