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Vladimir Hachinski, MD, FRCPC, DScMed
London, Ontario

Arch Neurol. 1998;55:742.

	Since this article does not have an abstract, we have provided the first 150 words of the full text and any section headings.

TEA BRIMS with aluminum. Although tea-drinking countries have idiosyncrasies, there is no evidence that they have a higher incidence of Alzheimer disease (AD). Despite its waning sway, the aluminum hypothesis associated with AD offered one of the first scientifically plausible explanations for the development of a classic neurodegenerative disease, yielding considerable understanding about aluminum neurotoxicity and the pathologic features of the brain.

Forbes and Hill review the evidence for an apparent association between aluminum exposure and the risk of developing AD. After acknowledging the methodological limitations of the studies cited, they answer the question with a tentative yes. Munoz counters with a resounding no and combines pathologic and epidemiological evidence to buttress his position. Moreover, he makes the point that a continued espousal of the aluminum hypothesis would be fruitless and harmful.

The time has come to take a stand against the periodic panics that . . . [Full Text of this Article]

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