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Morvarid Karimi, MD; Mehrdad Razavi, MD; Deema Fattal, MD

Arch Neurol. 2004;61:583-585.

Background  Neurologic deficits in patients with Klippel-Feil syndrome usually are attributed to direct compression of neuronal structures or hypoperfusion secondary to compression of the vertebral arteries by bony abnormalities.

Objective  To describe a 38-year-old woman with known Klippel-Feil syndrome who developed lateropulsion.

Results  The results of magnetic resonance imaging were consistent with rubrothalamic stroke. The cerebral angiogram confirmed vertebral artery dissection at the level of her previously observed bony abnormality.

Conclusions  Hypermobility adjacent to fused vertebrae subjects the vertebral artery to increased shear forces. Thus, Klippel-Feil syndrome may be a predisposing factor for vertebral artery dissection. Moreover, to our knowledge, this case represents the second known case of rubral lateropulsion.

From the Department of Neurology, University of Iowa Hospitals and Clinics, Iowa City.

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