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Allan H. Ropper, MD; Lester Adelman, MD

Arch Neurol. 1992;49(9):979-981.

Abstract
• A patient with typical acute Guillain-Barré syndrome died 72 hours after his first symptoms occurred, and an autopsy was performed 8 hours after his death. Extensive sampling of cranial and peripheral nerves, sensory ganglia, and autonomic nerves showed only minimal inflammatory lymphocytic and macrophage infiltrates. This case, one of the earliest studied extensively, represents an extreme example of a noninflammatory mechanism that has been proposed in some cases of Gullain-Barré syndrome.

Author Affiliations
From the Division of Neurology, St Elizabeth's Hospital, and the Neuropathology Laboratory, Tufts University School of Medicine, Boston, Mass.

Footnotes
Accepted for publication March 31, 1992.

Reprint requests to Neurology Service, St Elizabeth's Hospital, 736 Cambridge St, Boston, MA 02135 (Dr Ropper).

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