This Article  • References  • Full text PDF  • Reply to article  • Send to a friend  • Save in My Folder  • Save to citation manager  • Permissions  Citing Articles  • Citation map  • Citing articles on HighWire  • Contact me when this article is cited  Related Content  • Similar articles in this journal  Social Bookmarking   What's this?

Ariyuki Hori, MD; Genjiro Hirose, MD; Satoshi Kataoka, MD; Katsuyuki Tsukada, MD; Kei Furui, MD; Hisao Tonami, MD

Arch Neurol. 1991;48(8):871-874.

Abstract
• A 13-year-old boy was the victim of attempted strangulation. His condition had returned to normal by the sixth day after the assault; however, from the seventh day, choreoathetosis, dystonia, and marked pseudobulbar paralysis developed in the boy. The computed tomographic scans and T₂-weighted magnetic resonance images that were obtained at this time revealed lowdensity and high-signal intensities in the region of the bilateral putamen and caudate nucleus. These symptoms and the changes in his computed tomographic scans and magnetic resonance images subsided gradually during a 2-month period. Sequential analysis of the cerebrospinal fluid for -aminobutyric acid and dopamine concentrations during his illness revealed reciprocal changes with normal recovery. Because of the delayed onset of neurological changes and the cerebrospinal fluid showing reversible symptoms, the delayed encephalopathy after strangulation had been related to the biochemical alterations that followed anoxia in the vulnerable basal ganglia.

Author Affiliations
From the Departments of Neurology (Drs Hori, Hirose, Kataoka, Tsukada, and Furui) and Radiology (Dr Tonami), Kanazawa Medical University, Ishikawa, Japan.

Footnotes
Accepted for publication December 21, 1990.

Presented at the First International Stroke Congress, Kyoto, Japan, October 16,1989.

Reprint requests to Department of Neurology, Kanazawa Medical University, Uchinada, Kahoku, Ishikawa Prefecture, Japan 920-02 (Dr Hori).

CiteULike    Connotea    Del.icio.us    Digg    Reddit    Technorati    Twitter     What's this?

THIS ARTICLE HAS BEEN CITED BY OTHER ARTICLES

Postanoxic parkinsonism: Clinical, radiologic, and pathologic correlation
Li et al.
Neurology 2000;55:591-593.
ABSTRACT | FULL TEXT  

Chasing the dragon - neurological toxicity associated with inhalation of heroin vapour: case report
Hill et al.
CMAJ 2000;162:236-238.
FULL TEXT  

Delayed Postanoxic Encephalopathy: Possible Role for Apoptosis
Sheth and Bodensteiner
J Child Neurol 1998;13:347-348.
 

Hypoxia-Induced CHAP Syndrome
Gingold et al.
J Child Neurol 1995;10:70-72.