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The Case of Conduction Aphasia

Daniel Kempler, PhD; E. Jeffrey Metter, MD; Catherine A. Jackson, MA; Wayne R. Hanson, PhD; Walter H. Riege, PhD; John C. Mazziotta, MD, PhD; Michael E. Phelps, PhD

Arch Neurol. 1988;45(3):275-279.

Abstract
• Ten patients with conduction aphasia were studied with computed tomography and 18-F-fluorodeoxyglucose positron emission tomography to examine glucose metabolism. Computed tomographic results identified a postrolandic structural locus for conduction aphasia. All patients demonstrated resting glucose hypometabolism throughout the parietal and temporal regions, and half of the patients also demonstrated reduced metabolic rates in the posterior, inferior, frontal (Broca's) regions. These data suggest that disconnection between posterior and anterior language areas may not be the best anatomical explanation for conduction aphasia.

Author Affiliations
From the School of Gerontology and School of Medicine, Department of Otolaryngology, University of Southern California, Los Angeles (Dr Kempler); the Veterans Administration Medical Center, Sepulveda, Calif (Drs Kempler, Metter, Hanson, and Riege, and Ms Jackson); Department of Neurology (Drs Metter and Mazziotta), Division of Nuclear Medicine and Biophysics (Drs Kempler, Metter, Riege, Mazziotta, and Phelps, and Ms Jackson), the School of Medicine, UCLA.

Footnotes
Accepted for publication Oct 23, 1987.

Presented in part before the Clinical Aphasiology Conference, Jackson Hole, Wyo, June 9, 1986, and the American Neurologic Association, San Francisco, October 1987.

Reprint requests to Otolaryngology/Speech Pathology (OPD 2P52), Los Angeles County/USC Medical Center, 1175 Cummings St, Los Angeles, CA 90033 (Dr Kempler).

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