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Delayed Traumatic Midbrain SyrinxClinical, Pathologic, and Electrophysiologic Features
David C. Anderson, MD;
Scott Bundlie, MD;
David A. Larson, MD;
Gaylan Rockswold, MD;
Angeline Mastri, MD
Arch Neurol. 1988;45(2):221-225.
Abstract
Seven months following severe crushing closed-head trauma with initial excellent recovery, neurologic deficits referable to the right mesencephalon abruptly developed in a 28-year-old man. Computed tomography demonstrated a cystic midbrain lesion with apparent communication with the aqueduct (later confirmed at autopsy). Brain-stem auditory evoked potentials after the clinical deterioration showed depression of amplitude of wave V, compared with predeficit records, only when the ear contralateral to the brain-stem lesion was stimulated.
Author Affiliations
From the Departments of Neurology (Drs Anderson, Bundlie, Larson, and Mastri) and Neurosurgery (Dr Rockswold), Hennepin County Medical Center and University of Minnesota Medical School, Minneapolis.
Footnotes
Accepted for publication July 8, 1987.
Reprint requests to the Department of Neurology, Hennepin County Medical Center, 701 Park Ave S, Minneapolis, MN 55415 (Dr Anderson).
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