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José M. Ferro, MD; Andrew Kertesz, MD, FRCP(C)

Arch Neurol. 1984;41(4):422-424.

Abstract
• A persistent hemispatial neglect developed in a patient following a small infarction of the posterior limb of the right internal capsule. This defect was restricted to tasks requiring spontaneous motor performance on the contralateral hemispace, especially when carried out under visual control. It was not associated with an extinction phenomenon. Verbal cues improved exploratory behavior, suggesting that hemispatial akinesia in this case was due to an intentional disorder. A pulvinar-parietal disconnection, depriving the right parietal cortex from relevant visuospatial information from the contralateral hemispace, was probably the anatomic basis of hemispatial neglect in this case.

Author Affiliations
From the Department of Clinical Neurological Sciences, University of Western Ontario, St Joseph's Hospital, London (Drs Ferro and Kertesz); and the Department of Neurology, Hospital de St Maria, Lisbon (Dr Ferro).

Footnotes
Accepted for publication Aug 13, 1983.

Reprint requests to Department of Clinical Neurological Sciences, University of Western Ontario, St Joseph's Hospital, London, Ontario, Canada N6A 4V2 (Dr Kertesz).

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