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Robert S. Baker, MD, FRCP(C); J. Raymond Buncic, MD, FRCS(C)

Arch Neurol. 1984;41(12):1274-1276.

Abstract
• A patient with a 16-month history of papilledema from pseudotumor cerebri had sudden loss of vision in one eye because of a central retinal artery occlusion (CRAO). The CRAO was transient and visual acuity improved from light perception to 6/7.5 OS in long-term follow-up. A visual field abnormality and color-vision deficit have persisted. Both eyes demonstrated peripapillary neovascular membranes. The eye with CRAO had extensive chorioretinal folds extending from the disc through the macula. Thorough investigations failed to demonstrate a coagulation abnormality, hyperviscosity, or source of emboli. Although the mechanism remains unclear, we postulate that CRAO may have been caused by a local pathologic condition in the optic nerve.

Author Affiliations
From the Department of Ophthalmology, The Hospital for Sick Children, Toronto (Dr Buncic) and the Department of Ophthalmology, Chandler Medical Center, Lexington, Ky (Dr Baker).

Footnotes
Accepted for publication Oct 27, 1983.

Reprint requests to Department of Ophthalmology, N152 Chandler Medical Center, 800 Rose St, Lexington, KY 40536-0084 (Dr Baker).

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