You are seeing this message because your Web browser does not support basic Web standards. Find out more about why this message is appearing and what you can do to make your experience on this site better.


ABOUT ARCHIVES
Advanced Search

Welcome   | My Account | E-mail Alerts | Access Rights | Sign In


  Vol. 41 No. 11, November 1984 TABLE OF CONTENTS
  Archives
  •  Online Features
  ORIGINAL ARTICLES
 This Article
 •References
 •Full text PDF
 • Reply to article
 •Send to a friend
 • Save in My Folder
 •Save to citation manager
 •Permissions
 Citing Articles
 •Citation map
 •Citing articles on HighWire
 •Citing articles on Web of Science (46)
 •Contact me when this article is cited
 Related Content
 •Similar articles in this journal
 Social Bookmarking
  Add to CiteULike Add to Connotea Add to Del.icio.us Add to Digg Add to Reddit Add to Technorati Add to Twitter What's this?

Anticoagulation and Hemorrhagic Infarction in Cerebral Embolism Secondary to Rheumatic Heart Disease

Luis Calandre, MD; Juan Fernández Ortega, MD; Félix Bermejo, MD

Arch Neurol. 1984;41(11):1152-1154.


Abstract

• The immediate clinical course of 42 patients with cerebral embolism secondary to rheumatic heart disease (RHD) was analyzed retrospectively. All the cases included cranial computed tomographic (CT) findings. Twenty-five patients received early anticoagulant therapy (AT) and 17 did not. Recurrent embolic events occurred in only one case. Seven instances of hemorrhagic infarction (HI) were found. In five cases, the development of HI correlated with a recurrent stroke, which occurred in each case within 48 hours after the initial cerebral embolism. Three of the patients with HI died (all of them were receiving AT). We believe that HI is a frequent cause of recurrent stroke after a cerebral embolic event, that it generally occurs in the first 48 hours, and that its outcome is possibly worsened by AT. Anticoagulant therapy should be delayed for three days after a cerebral embolic event secondary to RHD.



Author Affiliations

From the Department of Neurology, "1 de Octubre" University Hospital, Madrid.


Footnotes

Accepted for publication July 14, 1983.

Reprint requests to Servicio de Neurologia, Hospital "1 de Octubre," Ctra de Andalucia, Madrid, Spain (Dr Calandre).



Add to CiteULike CiteULike   Add to Connotea Connotea   Add to Del.icio.us Del.icio.us   Add to Digg Digg   Add to Reddit Reddit   Add to Technorati Technorati   Add to Twitter Twitter     What's this?

THIS ARTICLE HAS BEEN CITED BY OTHER ARTICLES

Prediction of Impending Hemorrhagic Transformation in Ischemic Stroke Using Magnetic Resonance Imaging in Rats • Editorial Comment
Knight et al.
Stroke 1998;29:144-151.
ABSTRACT | FULL TEXT  

Intracerebral Hemorrhage After Experimental Embolic Infarction: Anticoagulation
Lyden et al.
Arch Neurol 1987;44:848-850.
ABSTRACT  

Cardioembolic Stroke, Early Anticoagulation, and Brain Hemorrhage
Cerebral Embolism Study Group
Arch Intern Med 1987;147:636-640.
ABSTRACT  

Cardiogenic Brain Embolism
Cerebral Embolism Task Force
Arch Neurol 1986;43:71-84.
ABSTRACT  

Early Recurrent Embolism Rheumatic Heart Disease
Hart
Arch Neurol 1985;42:1033-1033.
ABSTRACT  

Anticoagulation and Bleeding Into Embolic Infarcts
Bogousslavsky
Arch Neurol 1985;42:1033-1034.
ABSTRACT  

Cere bral Emboli of Cardiac Origin
Hart
Arch Intern Med 1985;145:1329-1329.
ABSTRACT  





HOME | CURRENT ISSUE | PAST ISSUES | TOPIC COLLECTIONS | CME | SUBMIT | SUBSCRIBE | HELP
CONDITIONS OF USE | PRIVACY POLICY | CONTACT US | SITE MAP
 
© 1984 American Medical Association. All Rights Reserved.