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Delayed Hemorrhagic InfarctionA Cause of Late Neonatal Germinal Matrix and Intraventricular Hemorrhage
Laura R. Ment, MD;
Richard A. Ehrenkranz, MD;
Charles C. Duncan, MD;
Robert C. Lange, PhD
Arch Neurol. 1984;41(10):1036-1039.
Abstract
• Although a single perinatal or postnatal event may be directly correlated to intraventricular hemorrhage (IVH) in some infants, in other infants IVH may be related to a series of insults. Asphyxia, hypotension, and a pressure-passive low cerebral blood flow (CBF) may lead to an infarction. Subsequent events known to cause sudden rises in the CBF may then produce a hemorrhage into damaged tissues. We report two cases of this proposed model for delayed hemorrhage into infarcted tissues, or late IVH. Both neonates were severely asphyxiated, and both experienced profound hypotension and a low CBF on the first postnatal day. Late IVH was found in both neonates; at 2 to 3 months of age, one neonate was found to have computed tomographic evidence for diffuse encephalomalacia, and the other neonate was noted to have an occipital porencephalic cyst.
Author Affiliations
From the Departments of Pediatrics (Drs Ment, Ehrenkranz, and Duncan), Neurology (Dr Ment), Neurosurgery (Dr Duncan), Obstetrics and Gynecology (Dr Ehrenkranz), and Diagnostic Radiology (Dr Lange), Yale University School of Medicine, New Haven, Conn.
Footnotes
Accepted for publication Nov 18, 1983.
Reprint requests to the Department of Pediatrics, Yale University School of Medicine, 333 Cedar St, New Haven, CT 06510 (Dr Ment).
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