This Article  • References  • Full text PDF  • Reply to article  • Send to a friend  • Save in My Folder  • Save to citation manager  • Permissions  Citing Articles  • Citing articles on HighWire  • Citing articles on Web of Science (2)  • Contact me when this article is cited  Related Content  • Similar articles in this journal  Social Bookmarking   What's this?

LCDR Michael W. Devereaux, MC, USNR; Michael J. Partnow, MD

Arch Neurol. 1975;32(10):704-705.

Abstract
• Three days after an episode of hypoxia, a 20-year-old man developed profound motor deficit in the absence of behavioral or cognitive disturbance. Previous reviews of delayed hypoxic encephalopathy have stressed behavioral and cognitive disturbances as the initial symptoms. This patient's pyramidal tract dysfunction in the absence of higher cortical dysfunction serves to illustrate that delayed hypoxic encephalopathy is predominantly a white matter rather than a gray matter disorder.

Author Affiliations
From the Section of Neurology, Department of Medicine, Navy Regional Medical Center, Philadelphia, the Division of Neurology, Hahnemann Medical College, Philadelphia, and the departments of neurology of Rancocas Valley Hospital, Willingboro, NJ, and the University of Pennsylvania.

Footnotes
Accepted for publication Jan 7, 1975.

Reprint requests to Department of Neurology, Rancocas Valley Hospital, Willingboro, NJ 08046 (Dr. Partnow).

CiteULike    Connotea    Del.icio.us    Digg    Reddit    Technorati    Twitter     What's this?

THIS ARTICLE HAS BEEN CITED BY OTHER ARTICLES

Delayed Postanoxic Encephalopathy: Possible Role for Apoptosis
Sheth and Bodensteiner
J Child Neurol 1998;13:347-348.