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Robert N. Tyson, MD

Arch Neurol. 1974;30(5):409-411.

Abstract
A patient who received succinylcholine chloride developed the signs of cerebral death as a result of dysfunction of succinylcholine metabolism by the enzyme, pseudocholinesterase. However, the electroencephalogram was only moderately abnormal and the patient recovered fully in eight hours, emphasizing the importance of the EEG in the evaluation of cerebral death.

Author Affiliations
Seattle

From the Department of Medicine, Division of Neurology, University of Washington Hospital, Seattle.

Footnotes
Accepted for publication Dec 24, 1973.

Reprint requests to Auburn General Hospital, Auburn, WA 98002 (Dr. Tyson).

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