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Visual Agnosia-ProsopagnosiaA Clinicopathologic Correlation
D. Frank Benson, MD;
Joseph Segarra, MD;
Martin L. Albert, MD
Arch Neurol. 1974;30(4):307-310.
Abstract
Occlusive vascular disease of the posterior cerebral arteries produced infarction destroying the left medial occipital area, the splenium of the corpus callosum, and the right inferior longitudinal fasciculus of a patient with visual agnosia. Some of the reported neurobehavioral abnormalities (alexia without agraphia, color agnosia, and verbal memory defect) were readily correlated with the observed lesions. Other abnormalities, particularly the visual agnosia and prosopagnosia, are less common, but tentative anatomical correlations can be postulated. The involvement of the nondominant inferior longitudinal fasciculus would appear to be particularly meaningful.
Author Affiliations
Boston
From the Aphasia Research Center, Department of Neurology, Boston University School of Medicine, and the Neurobehavioral Center, Boston Veterans Administration Hospital, Boston.
Footnotes
Accepted for publication Oct 9, 1973.
Reprint requests to Department of Neurology, Boston Veterans Administration Hospital, 150 S Huntington Ave, Boston 02130 (Dr. Benson).
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