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Kathleen M. Foley, MD; H. Richard Beresford, MD

Arch Neurol. 1974;30(2):182-183.

Abstract
A 16-year-old boy developed an acute neurological illness characterized by flaccid paraplegia, bladder dysfunction, and striking sensory disturbances in the lower extremities. A myelogram was normal, but there was a pleocytosis and increased content of protein in the cerebrospinal fluid. Serologic and epidemiologic studies indicated a type I poliovirus infection. He had never received polio vaccine. Although previous reviews have stressed its rarity, this patient is described as an additional example of prominent sensory loss in acute poliomyelitis.

Author Affiliations
New York and Manhasset

From the Division of Neurology, Department of Medicine, North Shore Hospital, Manhasset, NY, and the Department of Neurology, Cornell University Medical College, New York.

Footnotes
Accepted for publication Aug 9, 1973.

Reprint requests to Division of Neurology, North Shore Hospital, Manhasset, NY 11030 (Dr. Beresford).

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THIS ARTICLE HAS BEEN CITED BY OTHER ARTICLES

Sensory Losses in Poliomyelitis
Seggey et al.
Arch Neurol 1976;33:664-664.
ABSTRACT