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Russell D. Snyder, MD

Arch Neurol. 1972;26(4):379-381.

Abstract
Three siblings were poisoned by the accidental ingestion of organic mercury and a fourth sibling, a newborn, was poisoned by the transplacental transfer of organic mercury. All developed prominent involuntary movement including resting tremor, ataxia, chorea, athetosis, and myoclonus. Previous descriptions of mercury poisoning, both organic and inorganic, have noted similar movement disorders. The pathogenesis of the involuntary movements is not entirely clear. One of the diagnostic considerations in a patient who acquires otherwise unexplained involuntary movements should be chronic mercury poisoning.

Author Affiliations
Albuquerque, NM

From the departments of pediatrics and neurology, University of New Mexico School of Medicine, Albuquerque, NM.

Footnotes
Accepted for publication Sept 15, 1971.

Reprint requests to University of New Mexico School of Medicine, 2211 Lomas Blvd NE, Albuquerque, NM 87106 (Dr. Snyder).

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THIS ARTICLE HAS BEEN CITED BY OTHER ARTICLES

Late EEG Findings and Clinical Status After Organic Mercury Poisoning
Brenner and Snyder
Arch Neurol 1980;37:282-284.
ABSTRACT