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David Burke, MB; Colin Andrews, MB, MRACP; Peter Ashby, MD, FRCP(C)

Arch Neurol. 1971;25(4):367-372.

Abstract
Autogenic inhibition of the H reflex was produced by static stretch of the triceps surae in nine patients with spasticity due to spinal cord lesions. This inhibitory effect was abolished by diffuse infiltration of the triceps surae with procaine hydrochloride, but enhanced by ischemia sufficient to block the ankle jerk without altering the direct motor response. In contrast to the triceps surae, the H reflex of the pretibial flexors was facilitated by stretch of these muscles. It is concluded that these effects are consistent with activation of secondary spindle endings which are known to produce autogenic inhibition of extensor reflexes and autogenic facilitation of flexor reflexes in spinal preparations.

Author Affiliations
Sydney, Australia

From the Division of Neurology, Prince Henry Hospital, and the School of Medicine, University of New South Wales, Sydney, Australia.

Footnotes
Accepted for publication March 25, 1971.

Reprint requests to Division of Neurology, Prince Henry Hospital, Little Bay, NSW 2036, Australia (Dr. Burke).

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